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Senate panel backs healthcare reform

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HEALTH_CARE_1078

Senate panel backs healthcare reform

A key U.S. Senate committee endorsed a sweeping healthcare overhaul on Tuesday, gaining the support of an influential Republican and delivering President Barack Obama a victory on his top domestic priority.

The Democratic-controlled Senate Finance Committee approved the measure on a 14-9 vote, with Senator Olympia Snowe becoming the first Republican in Congress to back a healthcare reform bill.

“Today we reached a critical milestone in our effort to reform our healthcare system,” Obama said after the vote, warning there were still big challenges ahead for healthcare reform.

The bill, the last of five pending health measures to clear a committee in Congress, will be merged with the Senate health panel’s version in the next few weeks for a full Senate debate and floor votes.

Snowe, who had been courted by Obama and his fellow Democrats, said she still had reservations about the overhaul and could not guarantee her continued support as it advances.

“My vote today is my vote today. It doesn’t forecast what my vote will be tomorrow,” Snowe said.

Health insurer companies stocks fell on fears reform was gaining steam and would hurt profits if it passed. The S&P Managed Health Care index of large health insurers was down 1.9 percent.

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Ageing heart can be prevented, say scientists

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Ageing heart can be prevented, say scientists

Scientists in Japan said they have uncovered evidence that shows it may be possible to delay or prevent heart failure in humans.

In a paper published in the journal Circulation, Tetsuo Shioi, lead researcher and assistant professor of medicine at Kyoto University Graduate School of Medicine in Kyoto, and his team described how they managed to suppress a variety of the P13K gene in a group of elderly mice.

The gene regulates the lifespan of cells and plays a role in the aging of tissues. In previous studies, suppression of this gene extended the lifespan of the roundworm and kept the hearts of old fruit flies healthy.

Compared with another group of mice in which the gene was left intact, mice with the suppressed gene had improved cardiac function, less fibrosis (which makes the heart inflexible) and fewer biological markers of aging.

“This study showed that aging of the heart can be prevented by modifying the function of insulin and paves the way to preventing age-associated susceptibility to heart failure,” Shioi said.

Old age is a major risk factor for heart failure, a condition when the heart is unable to pump enough blood around to supply the oxygen the body needs, the World Health Organization says.

According to the American Heart Association, 5.7 million Americans have heart failure, and nearly 10 out of every 1,000 people over age 65 suffer heart failure every year.

Mariell Jessup, professor of medicine at the University of Pennsylvania School of Medicine in Philadelphia, said older people experience a slow but gradual loss of heart cells and a host of other cellular abnormalities which make the remaining cells contract less efficiently.

“This early work in a mouse model, clarifying the role of PI3K in cardiac aging, could ultimately allow scientists to understand if human hearts are similarly influenced,” he said.

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Antioxidants may raise diabetes risk: study

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antioxidants

Antioxidants may raise diabetes risk: study

Instead of protecting against diabetes, antioxidants — compounds in foods and supplements that prevent cell damage — may actually increase the chances of getting diabetes, at least in the early stages, Australian researchers reported on Tuesday.

“In the case of early type 2 diabetes … our studies suggest that antioxidants would be bad for you,” Tony Tiganis of Monash University in Australia, whose study appears in the journal Cell Metabolism, said in a statement.

Antioxidants are protective proteins that can prevent cell damage caused by charged particles known as reactive oxygen species. This oxidative stress is thought to add to the progression of several diseases, including type 2 diabetes.

Because antioxidants fight oxidative stress, they have become a popular food supplement. But Tiganis said the picture appears to be a bit more complicated.

“We think there is a delicate balance, and that too much of a good thing — surprise, surprise — might be bad,” he said.

Tiganis’ team studied the effects of oxidative stress in mice fed a high-fat diet for 12 weeks. One group of mice lacked an enzyme known as Gpxl, which helps counter oxidative stress.

They found mice that lacked the enzyme were less likely to develop insulin resistance — an early sign of diabetes — than normal mice. But when they treated the enzyme-deficient mice with an antioxidant, “they lost this advantage and become more ‘diabetic,” Tiganis said in an e-mail.

He said oxidative stress may be working not to damage the body but to inhibit enzymes that hurt the body’s ability to use insulin early on in the development of diabetes, and that antioxidants remove this protective mechanism.

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